Osteopathy: The osteopathic lesion. Part 1

The osteopathic lesion. Part 1

We understand the suffering osteopathic lesion isolated from a joint or spinal segment of benign and reversible. Understanding where the characteristic signs are lack of mobility and pain.

This notion may be, in some cases, independent of anatomical and radiological state present the joint or spinal segment.

We may find a vertebra or more showing signs of osteoarthritis, in addition to the damaged discs, but can work perfectly and completely painless r no symptoms. In this case, although the radiological signs are being altered, no pain and mobility is sufficient, we will say that there osteopathic lesion, and therefore, there is nothing to discuss.

Clearly, might be a critique in the sense that the osteopathic lesion is not radiological translation. On the other hand would be curious that there were, as in the case of sprained fingers, so painful, not presents.

But they do not mean that the x-ray is not necessary. The study of the plate we offer valuable information and tell us about any contraindications that may exist.

It also sheds light on the state of spinal or other joints in our body and disc deterioration, advanced stages of osteoporosis, osteoarthritis, spinal alignment and lordosis, kyphosis or scoliosis, which at first sight not very apparent, and so on. But again, that this affected segment of OA or with your album this thin does not mean that pain is responsible for expressing the patient. So we could talk about the injury would require that these segments were looking doubtful sensitive radiological exploration. In this regard we would have three steps:

- Review of mobility
- Review and pain free joint
- State of the soft tissue.

In somatic dysfunction, radiological evidence questionable, we have to stop and study a neurophysiological phenomenon of vital importance: the fixing joint and muscle spasm.

The somatic dysfunction Nereo-muscular in nature, directly related to two types of receptors:

- The sensory receptors capsule-ligament.
- The spindles.

Recipients of the ligaments would cause pain if there is an abnormal stretching of the capsule-ligament system. This will also cause alterations of neuro-vascular origin in the same metamerism. The nociceptive stimuli increase the release of gamma motor neuron in the same area and resulted in causing muscle spasms. This muscle spasm is sustained physiological level as a result of an imbalance between intra-and extrafusal fibers, which prevents the muscle spindles to relax. This fixes certain muscle spasm joint segments.

Refunds when nothing was done to time and becomes chronic causes ossification of the tissues lose their natural texture and become fibrous, behaving as ligaments and further limits the field of natural joint movement.

To this also contributes to local neurovascular injury installed in the area. Tissue anoxia and pains promote tissue degeneration and the formation of edema. If all this happens in a vertebral segment, for instance have excessive responsiveness to nervous influences, therefore, will be a core area where the barriers of protection will be diminished.

This state of vulnerability and excitement can be extended to other neural areas located in spinal segment that innervates the injured joint. This condition can cause:

The myotome: muscle contractures.
The dermatome: Dermalgias and skin pain.
The sclerotome: Joint pain and ligament.
The enterotoma: Disorders of the visceral organs.

For more information visit our Osteopathy course online. Basic Concepts

*Automatic Translation